α-Glucosidase Activity Colorimetric Assay Kit
Cat.No. : | Kit-2202 |
Product Overview : | α-Glucosidase breaks down α-1,4 linked polysaccharides to glucose, which can be utilized as a source of energy.In the biotechnology industry, α-glucosidase is used to produce glucose from intermediate breakdown products of starch hydrolysis generated by enzymes such as amylase. Pompe disease, one of the 12 known glycogen storage diseases, is an autosomal recessive metabolic disorder attributed to α-glucosidase deficiency. In this disease, glycogen accumulates in the lysosomes, resulting in progressive muscle weakness, heart failure and other neurological symptoms. In α-Glucosidase Activity Colorimetric Assay Kit, α-Glucosidase hydrolyzes the Substrate Mix to release the p-nitrophenol that can be measured colorimetrically (OD = 410 nm). This is an easy, quick and high-throughput capable kit that can measure 0.1-10 mU of α-glucosidase activity in a variety of samples. |
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Applications : | Measurement of α-Glucosidase activity in biological samplesScreening α-Glucosidase inhibitors. |
Storage : | -20°C |
Size : | 100 assays |
For Research Use Only. Not intended for any clinical use. No products from Creative BioMart may be resold, modified for resale or used to manufacture commercial products without prior written approval from Creative BioMart.
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Q&As (20)
Ask a questionAGLU's role in glycogen breakdown helps maintain cellular glucose levels.
AGLU's activity provides glucose for energy needs in cells.
AGLU deficiency affects muscle cells due to glycogen accumulation.
AGLU's activity provides glucose for cellular energy production.
Yes, ongoing research explores AGLU's roles, implications, and potential therapies.
AGLU participates in metabolic pathways by breaking down glycogen.
AGLU has a specific protein structure that supports its enzymatic function.
Enzyme replacement therapy aims to restore AGLU's activity in Pompe disease.
AGLU's deficiency is targeted in Pompe disease treatments to restore enzyme activity.
Deficiency of AGLU causes Pompe disease, characterized by glycogen accumulation.
AGLU breaks down complex carbohydrates into simpler sugars like glucose.
Pompe disease results from AGLU deficiency, causing glycogen buildup and symptoms.
AGLU plays a key role in breaking down glycogen, releasing glucose for energy.
Yes, infantile-onset Pompe disease is characterized by severe AGLU deficiency.
Yes, AGLU's activity can be influenced by hormones and metabolic signals.
Yes, AGLU deficiency can affect liver health due to glycogen accumulation.
AGLU deficiency causes glycogen to accumulate, leading to muscle and organ issues.
AGLU deficiency leads to abnormal glycogen storage, causing tissue damage.
Yes, AGLU deficiency can lead to respiratory muscle weakness in Pompe disease.
Yes, AGLU deficiency affects heart health due to glycogen buildup.
Customer Reviews (5)
Write a reviewThe protein product’s consistent outcomes supported the reproducibility of our studies.
Compatibility across diverse assays expanded our research possibilities significantly.
AGLU interactions were effectively modulated by the protein product, broadening our understanding.
Employing the product across multiple cell types consistently yielded comparable results.
The product’s contributions to our insights justified its cost and return on investment.
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