• Official Full Name
  • RAB19, member RAS oncogene family
  • Synonyms
  • RAB19; RAB19, member RAS oncogene family; ras-related protein Rab-19; RAB19B; GTP-binding protein RAB19B; OTTHUMP00000212106; OTTHUMP00000212107
Source (Host):E. coliSpecies:Mouse
Product nameRecombinant Mouse RAB19, Member RAS Oncogene Family, GST-tagged
Source (Host):Mammalian CellsSpecies:Mouse
Product nameRecombinant Mouse RAB19 Protein
Source (Host):Mammalian CellsSpecies:Rat
Product nameRecombinant Rat RAB19 Protein
Source (Host):Mammalian CellsSpecies:Rhesus Macaque
Product nameRecombinant Rhesus monkey RAB19 Protein, His-tagged
Source (Host):Mammalian CellsSpecies:Chicken
Product nameRecombinant Chicken RAB19
Source (Host):Mammalian CellsSpecies:Chicken
Product nameRecombinant Chicken RAB19
Source (Host):Species:Human
Product nameRecombinant Human RAB19 293 Cell Lysate

Protein Function

Rab19 has several biochemical functions, for example, GTP binding, GTPase activity. Some of the functions are cooperated with other proteins, some of the functions could acted by Rab19 itself. We selected most functions Rab19 had, and list some proteins which have the same functions with Rab19. You can find most of the proteins on our site.
Related Protein
Function GTP binding
Related Protein TUBA3E; SAR1AB; GPN3; EFTUD2; TUBA1C; Arf2; DIRAS3; SUCLG2; ARF3B; GNL1
Function GTPase activity
Related Protein SEPT4; GIMAP7; LSG1; RAB39BB; RAB40C; RAB40A; MFN1B; RAB39BA; RAB11BA; GTPBP3

Interacting Protein

Rab19 has direct interactions with proteins and molecules. Those interactions were detected by several methods such as yeast two hybrid, co-IP, pull-down and so on. We selected proteins and molecules interacted with Rab19 here. Most of them are supplied by our site. Hope this information will be useful for your research of Rab19.

Rab19 Related Articles

Gillingham, AK; Sinka, R; et al. Toward a Comprehensive Map of the Effectors of Rab GTPases. DEVELOPMENTAL CELL 31:358-373(2014).
Schluth-Bolard, C; Labalme, A; et al. Breakpoint mapping by next generation sequencing reveals causative gene disruption in patients carrying apparently balanced chromosome rearrangements with intellectual deficiency and/or congenital malformations. JOURNAL OF MEDICAL GENETICS 50:144-150(2013).