Recombinant Human TGFBR3 Protein, His (Fc)-Avi-tagged
Cat.No. : | TGFBR3-4488H |
Product Overview : | Recombinant Human TGFBR3 with His (Fc)-Avi tag was expressed and purified |
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Source : | HEK293 |
Species : | Human |
Tag : | His&Fc&Avi |
Endotoxin : | < 1.0 EU per μg of the protein as determined by the LAL method |
Purity : | ≥85% by SDS-PAGE |
Stability : | Stable for at least 6 months from the date of receipt of the product under proper storage and handling conditions. Avoid repeated freeze-thaw cycles. |
Storage : | For long term storage, aliquot and store at -20 to -80 centigrade. Avoid repeated freezing and thawing cycles. |
Storage Buffer : | PBS buffer |
Gene Name : | TGFBR3 transforming growth factor, beta receptor III [ Homo sapiens ] |
Synonyms : | TGFBR3; transforming growth factor, beta receptor III; BGCAN; betaglycan; betaglycan proteoglycan; transforming growth factor, beta receptor III (betaglycan, 300kDa); TGF-beta receptor type III; TGFR-3 |
Gene ID : | 7049 |
mRNA Refseq : | NM_003243 |
Protein Refseq : | NP_003234 |
MIM : | 600742 |
UniProt ID : | Q03167 |
Products Types
◆ Recombinant Protein | ||
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Tgfbr3-298R | Recombinant Rat Tgfbr3 Protein, His-tagged | +Inquiry |
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TGFBR3-193H | Active Recombinant Human TGFBR3 | +Inquiry |
◆ Lysates | ||
TGFBR3-1847MCL | Recombinant Mouse TGFBR3 cell lysate | +Inquiry |
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Not For Human Consumption!
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Customer Reviews (3)
Write a reviewGreat product !
Seamless delivery process—ordered, received, and ready for experiments.
Timely delivery is key, and this product arrived on schedule.
Q&As (5)
Ask a questionTGFBR3 mutations are associated with hereditary connective tissue disorders, impacting extracellular matrix homeostasis and contributing to conditions like Loeys-Dietz syndrome.
TGFBR3 plays a role in embryonic development, and dysregulation may lead to developmental abnormalities due to disrupted TGF-beta signaling.
TGFBR3 facilitates cell-ECM interactions by modulating TGF-beta signaling, influencing processes like cell adhesion and migration.
TGFBR3 acts as a co-receptor, enhancing the binding of TGF-beta ligands to TGF-beta receptors, and modulates downstream signaling events.
TGFBR3 mutations are implicated in cardiovascular diseases, affecting TGF-beta-mediated processes in vascular smooth muscle cells and cardiac tissues.
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