Recombinant Mouse ATP7B Protein, His (Fc)-Avi-tagged

Cat.No. : ATP7B-885M
Product Overview : Recombinant Mouse ATP7B with His (Fc)-Avi tag was expressed and purified
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Source : HEK293
Species : Mouse
Tag : His&Fc&Avi
Endotoxin : < 1.0 EU per μg of the protein as determined by the LAL method
Purity : ≥85% by SDS-PAGE
Stability : Stable for at least 6 months from the date of receipt of the product under proper storage and handling conditions. Avoid repeated freeze-thaw cycles.
Storage : For long term storage, aliquot and store at -20 to -80 centigrade. Avoid repeated freezing and thawing cycles.
Storage Buffer : PBS buffer
Gene Name : Atp7b ATPase, Cu++ transporting, beta polypeptide [ Mus musculus ]
Official Symbol : ATP7B
Gene ID : 11979
mRNA Refseq : NM_007511.2
Protein Refseq : NP_031537.2
UniProt ID : Q64446

For Research Use Only. Not intended for any clinical use. No products from Creative BioMart may be resold, modified for resale or used to manufacture commercial products without prior written approval from Creative BioMart.


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Customer Reviews (3)

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    Impressed with the product quality.


      The product exceeded expectations.


        This product consistently delivers accurate results in our experiments.

        Q&As (5)

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        How does the ATP7B protein contribute to copper transport within cells, and what cellular compartments are involved in this process? 01/14/2022

        ATP7B facilitates copper transport from the cytoplasm to the Golgi apparatus, where copper is incorporated into proteins. It also mediates copper export from cells when copper levels are elevated, preventing toxicity.

        How does ATP7B interact with other proteins involved in copper metabolism, and what are the implications of disruptions in these interactions? 02/22/2021

        ATP7B interacts with various proteins in the copper transport pathway. Disruptions in these interactions can lead to mislocalization of ATP7B and compromise its ability to regulate copper levels properly.

        How does ATP7B-mediated copper transport impact the activity of copper-dependent enzymes, and what are the broader implications for cellular function? 09/18/2020

        ATP7B ensures the proper supply of copper to enzymes involved in various cellular processes, including energy production and antioxidant defense. Dysregulation can disrupt these processes, affecting cell function.

        What is the role of ATP7B in copper excretion at the systemic level, and how does this relate to copper metabolism in the body? 05/30/2019

        ATP7B is crucial for copper excretion into bile, a process that contributes to systemic copper balance. Dysfunctional ATP7B results in impaired copper excretion, leading to the accumulation of copper in tissues.

        Can you explain the consequences of mutations in the ATP7B gene, particularly in relation to Wilson's disease? 11/05/2018

        Mutations in the ATP7B gene lead to impaired copper transport, causing copper accumulation in tissues. This is associated with Wilson's disease, a disorder characterized by copper toxicity, especially in the liver and brain.

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